张艳
个人信息Personal Information
副教授
性别:女
在职信息:在职
所在单位:基础医学院
入职时间:2006-07-16
扫描关注
- [1] 张杨. HDAC9-mediated epithelial cell cycle arrest in G2/M contributes to kidney fibrosis in male mice. NATURE COMMUNICATIONS, 2023.
- [2] 刘素侠. 靶向于端粒酶调控区反义RNA腺病毒载体的构建. 《山东大学学报》(医学版), 43, 667, 2005.
- [3] 崔晓阳. GPR87 promotes renal tubulointerstitial fibrosis by accelerating glycolysis and mitochondrial injury. Free Radical Biology & Medicine, 58, 2022.
- [4] . GPR97 deficiency ameliorates renal interstitial fibrosis in mouse hypertensive nephropathy. ACTA PHARMACOLOGICA SINICA, 2023.
- [5] 孙玉. JAML promotes acute kidney injury mainly through a macrophage-dependent mechanism.. JCI Insight, 2022.
- [6] 王姿颖. Diacylglycerol kinase epsilon protects against renal ischemia/reperfusion injury in mice through Krüppel-like factor 15/klotho pathway. RENAL FAILURE, 2022.
- [7] 王晓杰. GPR87 promotes renal tubulointerstitial fibrosis by accelerating glycolysis and mitochondrial injury. Free Radical Biology & Medicine, 189, 58, 2022.
- [8] 詹萍. Myeloid-derived growth factor deficiency exacerbates mitotic catastrophe of podocytes in glomerular disease. Kidney International, 102, 546, 2022.
- [9] 靖旭. Gene deficiency or pharmacological inhibition of PDCD4-mediated FGR signaling protects against acute kidney injury. ACTA PHARMACEUTICA SINICA B, 11, 394, 2021.
- [10] 苏泽玉. CLEC14A protects against podocyte injury in mice with adriamycin nephropathy. FASEB Journal, 35, 2021.
- [11] 付艺. Elevation of JAML Promotes Diabetic Kidney Disease by Modulating Podocyte Lipid Metabolism. CELL METABOLISM, 32, 2020.
- [12] 周亚滨 , 卢翌 , 周蒙 , 唐伟 , 易凡 , 王姿颖 , 王晓杰 , 刘敏 , 张艳 , 孙玉 and 周迪. Progranulin alleviates podocyte injury via regulating CAMKK/AMPK-mediated autophagy under diabetic conditions. JOURNAL OF MOLECULAR MEDICINE-JMM, 2019.
- [13] 易凡 , 唐伟 , 孙玉 , 王晓杰 , 王姿颖 , 魏欣冰 , 张艳 and 付艺. Therapeutic Potential of Progranulin in Hyperhomocysteinemia-Induced Cardiorenal Dysfunction. Hypertension, 69, 259, 2017.
- [14] 易凡 , 张艳 , 于晓 , 王姿颖 , 包婵婵 and 魏欣冰. Novel Role of NOD2 in Mediating Ca2+ Signaling: Evidence from NOD2-regulated Podocyte TRPC6 Channels in Hyperhomocysteinemia.. Hypertension, 62, 506, 2013.
- [15] 易凡 , 王姿颖 , 张艳 , 王琳琳 , 魏欣冰 and 张斌. Regulation of NADPH Oxidase Activity Is Associated with miRNA-25-Mediated NOX4 Expression in Experimental Diabetic Nephropathy. Am J Nephrol, 32, 581, 2010.
- [16] 王姿颖 , 易凡 , 张艳 , 魏欣冰 and 张岫美. NOX2 deficiency ameliorates cerebral injury through reduction of complexin II-mediated glutamate excitotoxicity in experimental stroke.. Free Radic Biol Med., 65, 942, 2013.
- [17] 易凡 , 张艳 , 魏欣冰 and 王姿颖. TIPE2 acts as a negative regulator linking NOD2 and inflammatory responses in myocardial ischemia/reperfusion injury. J Mol Med (Berl), 2015.
- [18] 易凡 , 张艳 , 丁华 , 魏欣冰 , 甄军晖 and 王姿颖. Expression and regulation of a novel identified TNFAIP8 family is associated with diabetic nephropathy. BBA-Molecular Basis of Disease, 1802, 1078, 2010.
- [19] 唐伟 , 卢翌 , 易凡 , 王姿颖 , 王晓杰 , 刘敏 , 张艳 and 孙玉. PGRN acts as a novel regulator of mitochondrial homeostasis by facilitating mitophagy and mitochondrial biogenesis to prevent podocyte injury in diabetic nephropathy. Cell Death & Disease, 2019.
- [20] 易凡 , 王琳琳 , 张斌 , 魏欣冰 , 王姿颖 , 张艳 , 陈琳 HDAC4/5-HMGB1 signalling mediated by NADPH oxidase activity contributes to cerebral ischaemia/reperfusion injury. Journal of Cellular and Molecular Medicine, 2013.